It’s the Diseases We Get As We Age That Kill Us | Breakthrough

researchers at the buck Institute in

researchers at the buck Institute in Northern California are looking for ways

Northern California are looking for ways

Northern California are looking for ways to prevent the many diseases associated

to prevent the many diseases associated

to prevent the many diseases associated with aging by slowing the aging process

with aging by slowing the aging process

with aging by slowing the aging process itself to discover drugs that will one

itself to discover drugs that will one

itself to discover drugs that will one day increase health and human beings

day increase health and human beings

day increase health and human beings researchers start much lower on the

researchers start much lower on the

researchers start much lower on the evolutionary ladder with single-cell

evolutionary ladder with single-cell

evolutionary ladder with single-cell organisms the nice thing about yeast is

organisms the nice thing about yeast is

organisms the nice thing about yeast is that we can keep billions of them in the

that we can keep billions of them in the

that we can keep billions of them in the laboratory very easily if we’re testing

laboratory very easily if we’re testing

laboratory very easily if we’re testing drugs we can look at 20 or 30 drugs in a

drugs we can look at 20 or 30 drugs in a

drugs we can look at 20 or 30 drugs in a single week so so fast if a chemical

single week so so fast if a chemical

single week so so fast if a chemical compound works in yeast they test it in

compound works in yeast they test it in

compound works in yeast they test it in more complex organisms conditions seen

more complex organisms conditions seen

more complex organisms conditions seen in aging animals like worms have

in aging animals like worms have

in aging animals like worms have surprising similarities to serious

surprising similarities to serious

surprising similarities to serious diseases seen in ageing humans we’ve

diseases seen in ageing humans we’ve

diseases seen in ageing humans we’ve shown recently that during normal aging

shown recently that during normal aging

shown recently that during normal aging in worms there is an accumulation of

in worms there is an accumulation of

in worms there is an accumulation of insoluble proteins and we believe this

insoluble proteins and we believe this

insoluble proteins and we believe this material is toxic and lo and behold

material is toxic and lo and behold

material is toxic and lo and behold that’s exactly what many people in the

that’s exactly what many people in the

that’s exactly what many people in the Alzheimer’s field have been thinking

Alzheimer’s field have been thinking

Alzheimer’s field have been thinking about for years the sticky stuff that we

about for years the sticky stuff that we

about for years the sticky stuff that we find in the brains of Alzheimer’s

find in the brains of Alzheimer’s

find in the brains of Alzheimer’s patients the compounds that extend

patients the compounds that extend

patients the compounds that extend lifespan also reduce Alzheimer’s disease

lifespan also reduce Alzheimer’s disease

lifespan also reduce Alzheimer’s disease proteins Parkinson’s disease proteins if

proteins Parkinson’s disease proteins if

proteins Parkinson’s disease proteins if you target an aging process and you slow

you target an aging process and you slow

you target an aging process and you slow down aging then you slow down all the

down aging then you slow down all the

down aging then you slow down all the diseases in the pathology of Aging as

diseases in the pathology of Aging as

diseases in the pathology of Aging as well